Trenbolone Enanthate

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Trenbolone Enanthate is a synthetic derivative of Dihydrotestosterone with a strong androgenic action that causes muscular density and hardness to grow, as well as a mild anabolic effect that establishes a positive nitrogen balance in humans and promotes protein synthesis. Because SP Trenbolone Enanthate is a Dihydrotestosterone derivative, Trenbolone does not aromatize at any dosage and hence cannot be turned into estrogen. As a result, water retention caused by estrogen is no longer present. Trenbolone Enanthate is the longer-acting enanthate version. Use Enanthate.

Adverse effects:

• Dermatological/integumental: oily skin, acne vulgaris, acne conglobata, seborrhea, stretch marks (caused by rapid muscle enlargement), hypertrichosis (excessive body hair growth), androgenic alopecia (pattern hair loss; scalp baldness), fluid retention/edema.
• Reproductive/endocrine: libido fluctuations, reversible infertility, hypogonadotropic hypogonadism.
• Male-specific: spontaneous erections, nocturnal emissions, priapism, erectile dysfunction, gynecomastia (mostly with aromatizable and hence estrogenic AAS), oligospermia/azoospermia, testicular atrophy, intratesticular leiomyosarcoma, prostate hypertrophy, prostate cancer.
• Female-specific symptoms include masculinization, permanent voice deepening, hirsutism (excessive facial/body hair development), menstrual disorders (e.g., anovulation, oligomenorrhea, amenorrhea, dysmenorrhea), clitoral enlargement, breast atrophy, uterine atrophy, and teratogenicity (in female fetuses).
• Child-specific: premature epiphyseal closure and small height in boys, delayed puberty, and contrasexual precocity in girls.
• Psychiatric/neurological symptoms include mood swings, irritability, aggression, violent conduct, impulsivity/recklessness, hypomania/mania, euphoria, depression, anxiety, dysphoria, suicidality, delusions, psychosis, withdrawal, dependence, neurotoxicity, and cognitive impairment.
• Musculoskeletal conditions include muscle hypertrophy, strains, tendon ruptures, and rhabdomyolysis.
• Dyslipidemia (e.g., increased LDL levels, decreased HDL levels, reduced apo-A1 levels), atherosclerosis, hypertension, left ventricular hypertrophy, cardiomyopathy, myocardial hypertrophy, polycythemia/erythrocytosis, arrhythmias, thrombosis
• Hepatic symptoms include increased liver function tests (AST, ALT, bilirubin, LDH, ALP), hepatotoxicity, jaundice, hepatic steatosis, hepatocellular adenoma, hepatocellular cancer, cholestasis, and peliosis hepatis. These symptoms are largely or entirely caused by 17α-alkylated AAS.
• Renal conditions include renal hypertrophy, nephropathy, acute renal failure (due to rhabdomyolysis), focal segmental glomerulosclerosis, and renal cell cancer.
• Other conditions include glucose intolerance, insulin resistance, and immunological dysfunction.